Hemifacial spasm due to non-ketotic hyperglycemia
نویسندگان
چکیده
منابع مشابه
Hemichorea-Hemiballism Secondary to Non-Ketotic Hyperglycemia
Non-ketotic hyperglycemia is an unusual and rare cause of hemichorea-hemiballismus. Correction of the hyperglycemia usually results in total resolution of the signs and symptoms. We present the case and medical imaging findings of a 66-year-old female who presented with steadily worsening choreiform and ballistic movements of the right upper and lower extremities over a 2-week period. Her serum...
متن کاملNon-ketotic hyperglycemia unmasks hemichorea
BACKGROUND Chorea can be caused by a variety of diseases, including neurodegenerative disorders, vascular events, toxic-metabolic states, and immunologic and infectious diseases. We describe a patient who presented with hemichorea as the initial manifestation of Diabetes Mellitus (DM) and responded partially to the glycemic control. CASE REPORT A 63-year-old, healthy Hispanic man with no prio...
متن کاملA Case Series of Hemichorea due to Non Ketotic- Hyperglycemia with Unique MRI Brain Finding
Hemi chorea generally occurs due to a stroke, neoplasm or granuloma in the contra lateral basal ganglia [1]. Metabolic derangements especially hyperglycemia can also cause focal neurological dysfunction like hemiparesis, focal seizure [2,3]. Recently there have been case reports of hemichorea reported due to non-ketotic hyperglycemia with characteristic MRI findings predominantly in the Asian p...
متن کاملCT and hemifacial spasm.
Forty-six patients with typical hemifacial spasm had CT. Thirty-eight (83%) were abnormal, including two with surgically documented tumors. Thirty-six had a characteristic dolichoectatic vertebrobasilar artery, with the convexity pointing to the side of the spasm in 92% of the scans. This study suggests that CT is worthwhile procedure in the evaluation of hemifacial spasm and that dolichoectati...
متن کاملHemifacial spasm and craniovertebral anomaly.
Two patients with congenital anomaly of the craniovertebral junction causing disabling hemifacial spasm (HFS) are presented. In one patient, complete cessation of the HFS occurred for a period of two years following simple bony decompression of the craniovertebral junction raising unanswered questions as to the exact pathogenesis of HFS. Eventually both patients required microvascular decompres...
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ژورنال
عنوان ژورنال: International Journal of Advanced Medical and Health Research
سال: 2014
ISSN: 2350-0298
DOI: 10.4103/2349-4220.148016